Ataxia is Greek and means "disorder." The term actually describes quite well what ataxia is all about. It is a disorder of the horse's normal movement and posture that is caused by damage to the central nervous system. Ataxia is therefore not lameness!
Depending on the location of the damage, ataxia in horses is divided into three categories. If the causes of the disease lie in the cerebrum, midbrain, or cerebellum, it is referred to as "cerebral ataxia." This type of ataxia can be caused by severe head injuries or the consequences of serious infectious diseases.
If the cerebellum is affected, it is referred to as "cerebellar ataxia." This type of damage is often congenital. The first symptoms can therefore be detected shortly after birth. Among Oldenburg and Arabian horses, a congenital cerebellar disorder known as "Purzelkrankheit" is well known. However, cerebellar ataxia can also be triggered by a severe head injury or infection. The cerebellum is the coordination center of movement and is responsible for muscle tone and balance. Damage to the cerebellum therefore disrupts normal movement. In this case, a movement disorder of the forehand is usually noticed first. This is accompanied by twitching in the head and neck area and severe balance disorders, with affected horses sometimes falling sideways or backwards.
Spinal ataxia is characterized by damage to the spinal cord caused by narrowing of the spinal canal. This can be caused by bone fractures, inflammation of the vertebral joints, contusions, or compressions. A special form of spinal ataxia is Wobbler syndrome, a movement disorder that occurs mainly in young horses, the cause of which was unclear for a long time. Research now agrees that this is a multifactorial disease. Genetics and nutrition, together with additional external factors (injuries, falls, etc.), have a particular influence on the development of the disease. In fast-growing, large horses in particular, growth disorders of the vertebral bodies and resulting inflammation of the small vertebral joints are partly responsible for narrowing of the spinal canal in the neck area and thus compression of the spinal cord.
All three forms of ataxia cause damage to nerve tissue, which is then unable to transmit impulses.
Basically, the location and extent of the damage determine the type and severity of the symptoms. At the onset of the disease, the owner usually notices that the horse's gait is somewhat unsteady or unsteady. The horse's coordination and movement are impaired, which is particularly noticeable in tight turns or when trying to turn the horse backwards. In the worst case, the horses may even roll backwards. Furthermore, stumbling, jerky movements of the limbs, and buckling and crossing of the hind legs may occur. Abrupt stopping from a trot or walk causes particular difficulties for ataxic horses and can serve as a test to detect ataxia. In most cases of spinal ataxia, the symptoms are more pronounced in the hindquarters than in the forequarters. Affected horses are usually in good general health. Depending on the severity, muscle wasting may occur in the affected spinal segments and limbs, as well as reduced skin reflexes in these areas.
A classification according to the severity of symptoms in spinal ataxia was established by Böhm in 1977. Grade I describes mild symptoms, grade II moderate symptoms, and grade III severe symptoms.
If you notice any unusual or abnormal movements in your horse, you should consult a veterinarian immediately. If ataxia is suspected, the affected horse must not be ridden under any circumstances, as uncoordinated movements can pose a serious risk to both horse and rider.
A detailed preliminary report and the animal's exact medical history are particularly important for the diagnosis. This is the only way for the veterinarian to narrow down the possible causes of the impaired movement. They will then carry out a thorough clinical and special neurological examination to check the horse's responsiveness and movement. Even if the symptoms are relatively clear, an X-ray examination of the cervical spine should also be performed. This allows bone fractures or joint changes to be identified.
Furthermore, contrast medium injected into the spinal canal can be used to highlight narrowing of the spinal cord in the subsequent X-ray image. However, this is a very complex examination procedure that is only offered by a few equine clinics in Germany.
Due to the various causes and types of ataxia, different treatment approaches are available, although a complete cure is not always possible. If the ataxia is caused by poisoning or an infectious disease, it can usually be successfully treated with the appropriate medication.
Depending on the severity and appearance of the ataxia, anti-inflammatory and decongestant medications may be used to reduce pressure on the spinal cord. However, since nerve fibers only regenerate to a limited extent, a complete cure is not always possible. If bone structures are responsible for narrowing the spinal canal, surgery may be necessary, but this must always be performed under general anesthesia in a clinic. It is not uncommon for the horse's mobility to be severely restricted after surgery. Therefore, discuss the benefits and risks of such an operation with your veterinarian on a case-by-case basis.
In addition to medication or surgery, movement and coordination training, physical therapy, and/or hydrotherapy can help rebuild muscles and thus contribute significantly to a successful recovery. However, owners should be aware that most of the treatments mentioned above take a very long time and that the horse will not be rideable, or only to a limited extent, during this time and possibly also afterwards.
The chances of recovery for a horse with ataxia depend on the severity and cause of the condition, as does the treatment. While cerebral and cerebellar ataxia can be successfully treated in most cases by treating the underlying disease, the prognosis for spinal ataxia with severe symptoms is poor.
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